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ISNR - 2017 ISNR Annual Conference
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Objective 1: Enhance the knowledge and clinical application of evidenced-based assessment, treatment, neuro-modulation, and neurofeedback training techniques.
By meeting the above objective my professional competence will increase because I have acquired new strategies to use in my practice.
By meeting the above objective my professional performance will improve because I should be able to implement the new strategies.
By meeting the above objective my patient outcomes should improve due to the implementation of newly-learned strategies.
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Objective 2: Promote research and engagement into the methodologies, practices and theoretical concepts of neuroscience, EEG operant conditional and biofeedback
By meeting the above objective my professional competence will increase because I have acquired new strategies to use in my practice.
By meeting the above objective my professional performance will improve because I should be able to implement the new strategies.
By meeting the above objective my patient outcomes should improve due to the implementation of newly-learned strategies.
Objective 3: Accurately identify clinical symptomology and implement ethically sound, skilled treatment to a wide variety of individuals
By meeting the above objective my professional competence will increase because I have acquired new strategies to use in my practice.
By meeting the above objective my professional performance will improve because I should be able to implement the new strategies.
By meeting the above objective my patient outcomes should improve due to the implementation of newly-learned strategies.
Name the various lobes of the brain 
Identify the unique functions of the separate lobes of the brain
Summarize the concepts of the Arousal Mode
Identify rapid diagnostic procedures
Interpret QEEG data
Identify common QEEG signatures for disorders
Point out EEG patterns for trauma
Determine conditions requiring full QEEG
Explain what defines the various montages and the circumstances of their use
Describe how LORETA and sLORETA-based interpretation and training provide unique capabilities compared to surface metrics
Identify the best subject inclusion and exclusion criteria for building a database
Tell the origins of the EEG
Identify t-tests, alpha and P levels, correlational relationships, and z-scores
Identify the origins of the ISF signal in cortex
State the behavior associated with Infraslow Frequencies in research
Identify equipment and software specific to slow wave training
Show how to master specific software and equipment used in ISF training
Implement ISF concepts in treatment populations
Name at least three prominent networks common across several models
Describe the hubs of the Default Mode Network
Identify the fundamental concept behind graph theory
Describe cross frequency communication in the context of networks adaptability
Devise a strategy for a network based protocol
Name basic principles of EEG preprocessing: artifact correction, automated spike detection, spectral analysis of EEG
Identify mechanisms of generation of infra-slow oscillations, EEG rhythms (theta, alpha, beta)
Explain the brain systems and mechanisms of their functioning
Summarize how to measure neuromarkers of normal information processing
Identify how to measure neuromarkers of abnormal information processing: ADHD, schizophrenia, OCD, autism
Describe how qeeg assessment data is combined with symptom and behavioral data to construct a brain-based intake
Explain how neurofeedback protocols that target specific EEG amplitude and connectivity parameters can be expected to lead to clinically significant functional change in clients
Explain how brain connectivity is reflected in QEEG measurements and in clinical presentation, and how changes in brain connectivity are reflected in client outcomes
Articulate the principles of sLORETA brain activation imaging and neurofeedback in clinical practice
Describe the principles of infra-slow fluctuations (ISF) and the literature base supporting its interpretation and use.
Tell the EEG Basics
Summarize how neurofeedback really works
Explain how to decide or target what to train
Explain the basic concepts needed for beginners
Summarize the different points of view in the field about the type of training
Explain the necessity of affect regulation in the treatment developmental trauma
Describe at least four protocols that can address fear and rage
Describe neurofeedback approaches to quieting shame
Describe the relationship between affect regulation and executive function.
Develop a plan for integrating neurofeedback and psychotherapy
Identify how pEMF, tDCS/tACS/tRNS affects the brain
Identify how stimulation modalities can enhance neurofeedback outcomes
Setup the NeuroField64 synchronized stimulation/neurofeedback platform
Utilize the NeuroField64 stimulation databases
Identify and quantify clinical symptoms in the
Explain the pathophysiological foundations of concussion
Identify QEEG patterns often found in concussion
Apply this knowledge to neurofeedback regiments to treat concussion
Summarize the experimental NF protocol for anxiety with active sham group condition
Identify additional qualitative and quantitative assessment tools for NF research
Identify methodological considerations for statistical NF data analysis
Summarize the generation of the EEG from a physiological point of view
Explain how clinically relevant brain function can be discerned using EEG and QEEG techniques
State how QEEG can be used to assess clients, and to assess client treatment progress
Distinguish distinct algorithms used to predict at least 3 basic cognitive functions in humans
Describe the relative importance of connectivity measures in predictive algorithms versus power measures of the EEG
Devise a strategy for a multivariate based protocol
Summarize ASD symptomology, diagnoses, and epidemiology
Explain the need for individualization of treatment
Use assessment information to inform the neurofeedback approach
Measure EEG phase reset in short distance inter-electrode combinations
Identify the ways of application of the methodology for predicting clinical outcome in response to pharmacological medication process for constructing and monitoring protocols of neuromodulation
Explain how a “Small World” mode of network dynamics is related to human intelligence
Summarize the theoretical basis of a newly developed methodology of extracting functional neuromarkers from spontaneous multi-channel EEG, event-related de/synchronization, and event-related potentials (ERP)
Identify the ways of application of the methodology for predicting clinical outcome in response to pharmacological medication
Identify the process for constructing and monitoring protocols of neuromodulation
Explain the generation of the EEG from a physiological point of view
Summarize how clinically relevant brain function can be discerned using EEG and QEEG techniques
State how QEEG can be used to assess clients, and to assess client treatment progress
Tell the differences between bivariate and multivariate coherence approaches
Identify various multivariate coherence assessment techniques
Describe the history of Deep States neurofeedback
Explain how research on psychedelic medicine relates to Deep States neurofeedback
Design neurofeedback protocols to promote a variety of Deep States
Identify the purpose of having and following professional ethics
Identify scope of practice issues related to qEEG and neurofeedback
Identify key aspects of client/patient orientation to qEEG and neurofeedback
Identify the level of anxiety and depression in clients based entirely on qEEG analysis
Determine the phase of client anxiety or depression based entirely on qEEG analysis
Deduce the best training locations for reducing symptoms of anxiety or depression based on qEEG analysis
List the multiple critical regulatory functions of astroglial cells
Demonstrate understanding of the action on the brain as well as the theoretical foundations for the ultra low frequency approach
Use Connectome maps of the brain’s interconnections to explain the spillover or global effect of Neurofeedback training to self and clients
Explain how EEG, QEEG and ERP are useful in the diagnosis of concussions and TBI 
Summarize how HRV is useful in reducing the physiological effects of TBI and can be combined with Neurofeedback
Talk about how Neurofeedback stimulation can be successful in treating the cognitive and neurological effects of TBI
Describe EEG collection and interpretation concepts, such as montages; active, reference, and ground electrodes, analog-to-digital conversion, QEEG, LORETA source density analysis
Name electrical and instrumentation terms including impedance, differential
Identify common artifacts including eye movement, muscle tension, cardiac, electrode movement
Recognize how cultural beliefs affect how trauma symptoms manifest
Recognize alternate symptoms of PTSD symptoms not usually seen in Western culture
Be aware how various barriers rooted in culture can prevent effective use of NF
Describe neurofeedback efficacy in symptom management of chemotherapy-induced peripheral neuropathy relative to other treatments
Describe current treatments for CIPN and results of current clinical trials
Describe effect sizes of each outcome measured in our trial, and implications for training protocols and future study design
1. Based on the content of this presentation, the participant will be able to understand the benefits of four channel neurofeedback for individuals with depressive symptoms
Based on the content of this presentation, the participant will be able to differentiate between the efficacy of various treatment options for depression
Based on the content of this presentation, the participant will be able to understand the extent to which neurofeedback will show maintained efficacy post treatment
. Based on the content of this presentation, the participant will be able to understand the epidemiology of traumatic brain injury and its subsequent clinical implications
Based on the content of this presentation, the participant will be able to understand how coherence measures and 4 channel multivariate neurofeedback relate to the reduction of symptomology associated with MTBI
Based on the content of this presentation, the participants will be able to understand the role time of treatment plays in the exploration of functional reductions of symptomology
Understand the importance of EEG data, and how it can reduce treatment failure
Identify the 3 most common causes for encephalopathy that they are likely to encounter in their practice
Identify the 4 neurobiomarkers associated with psychotropic medication failure
State the basic principle of Independent Component Analysis (ICA)
Describe at least 2 concerns for using ICA and other forms of auto-artifacting when artifacting the EEG
List at least 2 types of EEG epileptic events that may have clinical relevance
Explain how functional MRI works
Identify connectivity networks in the brain
State the transcutaneous vagus nerve stimulation targeting of specific brainstem centers
Summarize the use of SMR, Beta and Alpha/Theta protocols that are still being used
State how to choose protocols based on clinical data overlayed with knowledge of brain regions by function, including Brodmann areas
Review logic for choosing reward and inhibits frequencies
Summarize the development of early SMR, Beta and Alpha/Theta protocols that are still being used
Explain how to choose reward and inhibits
identify key components for protocol selections based on qEEGs
Explain how pulsed Electromagnetic (pEMF) frequencies can act as a cellular repair treatment
Explain how Infra Slow Oscillations (ISO) are related to glial cells that oversee key neuronal activities
Add Z-score cycling with ISO or pEMF to their practice providing they have the matching equipment and software combination
Define concussion injury
List at least one basic pathophysiologic principle of concussion injury at macroscopic, microscopic and molecular levels
Identify at least 5 signs and 5 symptoms of concussion injury
Describe the patterns of connectivity in the brain that underlie dissociation
Describe the role of affect regulation difficulties in developmental trauma
State a basic understanding of the connection between motherlessness, affect regulation and the sense of self
Identify which regions in the brain, indicated by current published research, are recruited when a person is experiencing heartbreak as a result of a disrupted romantic relationship
Evaluate the utility of qEEG assessment and sLORETA imaging in determining whether neurofeedback could potentially facilitate a post-relationship recovery arc for a client having difficulty resolving residual romantic feelings
Select regions to include in an sLORETA z-score neurofeedback training protocol intended to increase romantic resiliency
Describe the most common clinical epilepsy syndromes
Explain how the published data might inform choice of patient and specific protocol for seizure-focused neurofeedback training
Describe the common baseline findings on QEEG in patients with epilepsy
Name the QEEG subtypes of Autism and Asperger's and how they differ and overlap
Use QEEG and computerized testing to guide NF protocol selection and monitoring of NF results
Apply Neurofeedback strategies and techniques for Autism, Asperger's and ADD/ADHD
Identify how to reduce incorrect diagnosis in clinical setting
Explain bio markers
Describe how to improve children's academic successes
Explain how tDCS/tACS/tRNS and pEMF impacts the brain via BOLD MRI research
Summarize how tDCS/tACS/tRNs/pEMF stimulation methods can be synchronized with EEG neurofeedback to improve treatment outcomes
Learn from case presentations of actual clinical data
Summarize how the impact of developmental trauma on the brain functioning, well-being of the individual and society
List current research concerning the effects of neurofeedback on children ages 6-13 with developmental trauma
Summarize the role of neurofeedback in the treatment of treat children with developmental trauma
Recite the lifelong effects of Developmental Trauma and psychiatric affects
List the areas of the brain effected by developmental trauma
Summarize the brain function, coherence and connectivity issues associated with developmental trauma
Summarize the huge clinical/economical impact of a neurofeedback intervention for the treatment of multi-resistant chronic back pain
Discuss the Riemannian geometry and statistical blind source separation
List the requirements in terms of clinical trial methodology including post-hoc EEG data pre-processing and processing, statistical analysis, and correlation analysis
Understand that it is possible to evaluate and treat attention deficits in infants
Identify prenatal and perinatal risk factors for brain damage
Identify the most important white matter injuries
Name and discuss the function of key brain structures affected by childhood maltreatment
Discuss the concept of sensitive periods and how it applies to the effects of exposure to early life stress
Enumerate clinical and neurobiological differences between maltreated and non-maltreated individuals with the same primary psychiatric diagnosis
Summarize ASD symptomology, diagnoses, and epidemiology
Debate the need for individualization of treatment
Explain the potential causes and neuropsychological underpinnings of Autism Spectrum Disorder
Describe a phenomenological, or noetic, methodology to quantify consciousness (subjective experience) in reference to short stimulus conditions via retrospective phenomenological assessment (RPA)
Describe the methodological and statistical limits for using RPA to assess the intensities and patterns of consciousness via the Phenomenology of Consciousness Inventory (PCI)
Determine his or her own hypnotic responsivity index via a demonstrational/experiential exercise, illustrating how this approach can quantify phenomenological experience, in this case, in reference to an eyes closed period during hypnosis that each participant will experience
Describe the 4 different styles of meditation
Match client goals to specific meditation styles
Explain how each style fits with specific mental health concerns
Explain how to improve manual editing (artifacting QEEG data) skills
Point out differences between 4 databases (graphically)
Explain the function of several key ROI’s as well as the triple network
Describe the neurobiology of trauma and how it impacts upon symptom presentation
Identify the neural networks of trauma
Explain how to target commonly dysregulated neural networks PTSD with neurofeedback
Identify distinct ROI that can be selected appropriate for the types of behavior being explored
Describe the steps for data reduction of EEG variables that can be used when exploring which ROIs and what frequencies may be important to optimize to minimize a predisposition to have excessive anxiety. 
Devise a strategy for a multivariate-based protocol
Identify appropriate CPT codes for QEEG and Neurofeedback,
Identify and locate scope of practice for their profession in their state,
Name the different ways to bill insurance and considerations for deciding which way(s) to bill insurance
Demonstrate knowledge of learning theory and exposure therapy theory
Utilize Heart Rate Variability biofeedback in coping exposure
Design and compare Virtual Reality exposure protocols for phobic disorders
Identify the use of norms and age regression in enhancing sensitivity of QEEG
Identify EEG source localization as a surrogate for neuroimaging
State the importance of addressing the pathophysiological heterogeneity of disorders
Please rate the following:
The program was relevant to my work.
Content matched stated objectives.
Usefulness of handouts/AV.
Quality of facilities.
How well did the educational sessions give a balanced view of therapeutic options, including the use of generic names?
If you rated any of the above questions with 'fair,' 'poor,' 'disagree,' or 'strongly disagree' please explain in detail (e.g. session title, speaker name, situtation):
Do you believe this activity was appropriate for the scope of your professional activities?
Was the educational content scientifically sound?
If faculty spoke about off-label or investigational uses of a product, was that information disclosed to you?
Was the mode of education effective to learning?
If you answered "No" to any of the above questions, please explain.
Did you perceive any product/service/company/commercial bias in any educational session you attended or materials you received?
If you answered "Yes" to the above question, please detail the situation below (e.g. session title, speaker name):
Were you solicited by sales personnel in an educational room (other areas do not matter) while you attended this educational activity?
If you answered "Yes" to the above question, please explain in detail (e.g. who, when, where):
How much did you learn as a result of this education program?
What specifically did you learn during this activity that you intend to integrate into your practice?

What questions have arisen in your practice for which you need answers/strategies that you can implement?

What patient/client problems or patient/client challenges do you feel you are not able to address appropriately or to your satisfaction?
What problems are your patients/clients communicating to you that need attention or follow up?

Are you interested in basic, intermediate or advanced level trainings?

What barriers might you have that would interfere with implementation of new information learned from this training?

How can this training (the overall meeting) be improved to better impact competence, performance and/or patient/client outcomes?

Additional comments:

Only Florida licensees need to answer this question.  Notice to Florida Licensees:  In order for us to report your hours to CE Broker, you MUST provide us with your Florida alpha-numeric license number (e.g., CDCA.XXXXXX, LICDCII.XXXXXX, LCDCIII.XXXXXX, LICDC.XXXXXX).  Please be sure you enter this number accurately, or CE Broker will not record your hours.
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